GLP-1 Evidence

Not medical advice. A record of published research and our assessments of it. The limits

Metabolic Effects of a GLP-1 Analog Combined with Aerobic Training in Ovariectomized Rats

Design
Animal study · Unknown outcome
Match to healthy normal-weight adults aged 55–75
Not human evidence[Auto] Non-human (animal or cellular) evidence; no direct inference to any human population.
Could weight loss explain it?
Unknown[Auto] Non-human study; weight-loss mediation not assessable.
Study tier
Study tier 4[Auto] Preclinical evidence; not clinical evidence for any human population.
Assessment
Version 1 · automatic, not yet reviewed by a person · Sep 13, 2026

Study facts come from the paper. Population match, weight-loss explanation and study tier are our judgments, made against the reference group of healthy normal-weight adults aged 55–75.

[Auto, unreviewed; last sentences of abstract] This combined treatment reduced HDL levels, hepatic triglycerides and scWAT relative weight. These results suggest that SEM and EXE exert complementary effects on glycemic and lipid metabolism, with partially distinct metabolic outcomes that may involve different underlying mechanisms.

01Findings

What the study reported

Drugs
Semaglutide
Route
subcutaneous
Primary outcome
Not extracted
Effect
Not extracted
95% confidence interval
Not extracted
Follow-up
Not stated
Adverse events
Not extracted
Limitations
Auto-classified from abstract only; effect estimates, adverse events and limitations not extracted. Requires manual review.

Who was studied

Obesity status
obesity/overweight present (all or most)
Diabetes status
diabetes mentioned

Study quality details

Study design
Preclinical (animal)
Sample size
not extracted
Randomization
no
Blinding
not stated
Comparator
not stated
Follow up duration
not stated
Outcome type
unknown
Replication
not assessed (auto)
Consistency with other evidence
not assessed (auto)
Population applicability
VERY_INDIRECT
Statistical precision
not extracted
Risk of bias
not assessed (auto)
Funding conflicts
unclear
Peer review status
yes
02Funding

Funding and conflicts

Funding
not reported in abstract
Industry funded
Unclear
Manufacturer
None identified
Sponsor role
not reported in abstract
Author conflicts
Declaration of Competing Interest ☒ The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.
Independent replication
unknown

Funding is shown on every study and never used to score it.

04Source

The source, as retrieved

Abstract

Menopause is associated with visceral obesity, low-grade chronic inflammation, and increased risk of dyslipidemia, insulin resistance, and type 2 diabetes. Although exercise (EXE) and GLP-1 receptor agonists, such as semaglutide (SEM), have demonstrated beneficial effects on metabolic regulation, their combined impact in menopause models has been less investigated. In this study, ovariectomized female Wistar rats were divided into four experimental groups: sedentary plus saline (SED + SAL), sedentary plus semaglutide (SED + SEM), exercise (EXE + SAL), and EXE + SEM. The aerobic exercise protocol consisted of treadmill running at moderate intensity for four weeks, during which the rats received either SEM or SAL weekly. At the end of the protocol, metabolic parameters were assessed in blood, liver, soleus and gastrocnemius muscles. Both SEM groups had lower body weight, weight gain, relative subcutaneous white adipose tissue (scWAT) mass, glucose, total proteins, and triglycerides in serum and liver, while LDL cholesterol, glycogen in liver and gastrocnemius, and AMPK and HSF1 expression in gastrocnemius increased. EXE decreased total and LDL cholesterol, liver triglycerides and HSF1 expression in both tissues, while glycogen in liver and gastrocnemius, liver HSF1 expression and relative mass were increased. The combination of SEM + EXE increased glycogen content in the gastrocnemius and AMPK expression in both gastrocnemius and liver. This combined treatment reduced HDL levels, hepatic triglycerides and scWAT relative weight. These results suggest that SEM and EXE exert complementary effects on glycemic and lipid metabolism, with partially distinct metabolic outcomes that may involve different underlying mechanisms.

Where this record came from

SourceRetrievedIdentifier
pubmedSep 13, 202642727623
first ingestion